01.02.2014, 14:31
Hi
http://www.ilads.org/ilads_news/2012/rel...e-disease/
Verstehe ich das richtig das Bo Toxien macht Lpa ?
Wenn man die Westerblots anschaut bzw Banden kommt auch öfters das Wort Lippoproteine vor ein Zufall
Wenn ich es richtig verstehe sollte LPA doch ein Direknachweiss sein, der nicht sein darf oder ?
Auszug
The complex genetic sophistication of B. burgdorferiallows it to adapt to multiple environments and disseminate and evade the host’s immune system.4 In a compromised environment, B. burgdorferican survive by converting from a spirochetal form to granular and cys¬tic forms. After entry, B. burgdorferiactivates local inflammation yet evades host defenses and facilitates dissemina¬tion by potentially masquerading with host components such as plasmin and complement.5
B. burgdorferiis recognized by im¬mune cells and attacked by complement and antibodies. B. burgdorferiresponds by down-regulating its surface proteins and hiding in the extracellular matrix.6
Some of the psychiatric sequelae may be associated with direct effects, such as possible toxin release and cell pen¬etration and lysis. Like other infections, most symptoms are instead associated with immune effects. These immune effects include persistent inflammation with cytokine effects, the release of proinflammatory lipoproteins from the outer coat of B. burgdorferiand autoim¬munity
Hausel
http://www.ilads.org/ilads_news/2012/rel...e-disease/
Verstehe ich das richtig das Bo Toxien macht Lpa ?
Wenn man die Westerblots anschaut bzw Banden kommt auch öfters das Wort Lippoproteine vor ein Zufall
Wenn ich es richtig verstehe sollte LPA doch ein Direknachweiss sein, der nicht sein darf oder ?
Auszug
The complex genetic sophistication of B. burgdorferiallows it to adapt to multiple environments and disseminate and evade the host’s immune system.4 In a compromised environment, B. burgdorferican survive by converting from a spirochetal form to granular and cys¬tic forms. After entry, B. burgdorferiactivates local inflammation yet evades host defenses and facilitates dissemina¬tion by potentially masquerading with host components such as plasmin and complement.5
B. burgdorferiis recognized by im¬mune cells and attacked by complement and antibodies. B. burgdorferiresponds by down-regulating its surface proteins and hiding in the extracellular matrix.6
Some of the psychiatric sequelae may be associated with direct effects, such as possible toxin release and cell pen¬etration and lysis. Like other infections, most symptoms are instead associated with immune effects. These immune effects include persistent inflammation with cytokine effects, the release of proinflammatory lipoproteins from the outer coat of B. burgdorferiand autoim¬munity
Hausel